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anti cancer metabolism compounds library  (TargetMol)


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    TargetMol anti cancer metabolism compounds library
    Anti Cancer Metabolism Compounds Library, supplied by TargetMol, used in various techniques. Bioz Stars score: 94/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+cancer+metabolism+compound+library/Adrenergic+Receptor-Targeted+Compound+Library/pm41506347-317-5-12
    Average 94 stars, based on 6 article reviews
    anti cancer metabolism compounds library - by Bioz Stars, 2026-09
    94/100 stars

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    Drug discovery:

    Article Title: Potential role of lipophagy impairment for anticancer effects of glycolysis-suppressed pancreatic ductal adenocarcinoma cells.
    Article Snippet: .. An Anti-Cancer Metabolism Compound library (96-well, catalog number L2130) was purchased from TargetMol (Wellesley Hills, MA, USA). .. PANC-1 cells were cultured in glucose (2 g/L), galactose (2 g/L), or low-glucose (0.2 g/L) medium with 130 different types of metabolic compounds for 48 h. To assess the cell number, the cells were incubated with 0.5mg/mL 3-(4,5-dimethylthiazol-2-yl)- Cell Death Discovery (2024) 10:166 2,5-diphenyltetrazolium bromide (MTT; Dojindo Laboratory, Kumamoto, Japan) for 2 h. After incubation, the formazan crystals produced were dissolved in 200 μL DMSO.

    Article Title: Deficiency of SDHC promotes metastasis by reprogramming fatty acid metabolism in colorectal cancer
    Article Snippet: .. Anti-cancer Metabolism Compound Library containing 237 anti-cancer metabolism compounds was purchased from TargetMol(L2130). ..

    Article Title: Effects of SP-2509 and OG-L002 on lipophagy using target or off-target molecules in glycolysis-suppressed pancreatic ductal adenocarcinoma cells
    Article Snippet: .. Metabolic compound screening An Anti-Cancer Metabolism Compound library (96-well, catalog number L2130) was purchased from TargetMol (Wellesley Hills, MA, USA). .. PANC-1 cells were cultured in glucose (2 g/L), galactose (2 g/L), or low-glucose (0.2 g/L) medium with 130 different types of metabolic compounds for 48 h. To assess cell viability, the cells were then incubated with 0.5 mg/mL 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT; Dojindo Laboratory, Kumamoto, Japan) for 2 h. After incubation, the formazan crystals produced were dissolved in 200 μL DMSO.

    Article Title: Deficiency of SDHC promotes metastasis by reprogramming fatty acid metabolism in colorectal cancer
    Article Snippet: Additionally, the AKT inhibitor MK-2206 (T1952, TargetMol) was employed at a concentration of 1 μmol/L to treat CRC cells for 48 h when necessary. .. Anti-cancer Metabolism Compound Library containing 237 anti-cancer metabolism compounds was purchased from TargetMol(L2130). ..

    Article Title: Potential role of lipophagy impairment for anticancer effects of glycolysis-suppressed pancreatic ductal adenocarcinoma cells
    Article Snippet: .. An Anti-Cancer Metabolism Compound library (96-well, catalog number L2130) was purchased from TargetMol (Wellesley Hills, MA, USA). .. PANC-1 cells were cultured in glucose (2 g/L), galactose (2 g/L), or low-glucose (0.2 g/L) medium with 130 different types of metabolic compounds for 48 h. To assess the cell number, the cells were incubated with 0.5 mg/mL 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT; Dojindo Laboratory, Kumamoto, Japan) for 2 h. After incubation, the formazan crystals produced were dissolved in 200 μL DMSO.



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    Bioinformatics analysis. a The volcano plot represents the DEGs. b GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in biological processes. c GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in cellular components. d GO annotations of the DEGs. The Bulb map presents the enrichment scores of the top 10 significantly enriched GO terms in molecular functions. e KEGG pathway enrichment analysis of DEGs. f Proteasome pathway enriched by GSEA. g Parkinson disease pathway enriched by GSEA. h Fatty acid <t>metabolism</t> pathway enriched by GSEA. i Oxidative phosphorylation pathway enriched by GSEA. j butanoate metabolism pathway enriched by GSEA. k Citrate cycle (TCA cycle) pathway enriched by GSEA. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001
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    Bioinformatics analysis. a The volcano plot represents the DEGs. b GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in biological processes. c GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in cellular components. d GO annotations of the DEGs. The Bulb map presents the enrichment scores of the top 10 significantly enriched GO terms in molecular functions. e KEGG pathway enrichment analysis of DEGs. f Proteasome pathway enriched by GSEA. g Parkinson disease pathway enriched by GSEA. h Fatty acid <t>metabolism</t> pathway enriched by GSEA. i Oxidative phosphorylation pathway enriched by GSEA. j butanoate metabolism pathway enriched by GSEA. k Citrate cycle (TCA cycle) pathway enriched by GSEA. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001
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    Bioinformatics analysis. a The volcano plot represents the DEGs. b GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in biological processes. c GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in cellular components. d GO annotations of the DEGs. The Bulb map presents the enrichment scores of the top 10 significantly enriched GO terms in molecular functions. e KEGG pathway enrichment analysis of DEGs. f Proteasome pathway enriched by GSEA. g Parkinson disease pathway enriched by GSEA. h Fatty acid metabolism pathway enriched by GSEA. i Oxidative phosphorylation pathway enriched by GSEA. j butanoate metabolism pathway enriched by GSEA. k Citrate cycle (TCA cycle) pathway enriched by GSEA. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001

    Journal: Journal of Translational Medicine

    Article Title: Deficiency of SDHC promotes metastasis by reprogramming fatty acid metabolism in colorectal cancer

    doi: 10.1186/s12967-024-05361-x

    Figure Lengend Snippet: Bioinformatics analysis. a The volcano plot represents the DEGs. b GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in biological processes. c GO annotations of the DEGs. The Bulb map presents enrichment scores of the top 10 significantly enriched GO terms in cellular components. d GO annotations of the DEGs. The Bulb map presents the enrichment scores of the top 10 significantly enriched GO terms in molecular functions. e KEGG pathway enrichment analysis of DEGs. f Proteasome pathway enriched by GSEA. g Parkinson disease pathway enriched by GSEA. h Fatty acid metabolism pathway enriched by GSEA. i Oxidative phosphorylation pathway enriched by GSEA. j butanoate metabolism pathway enriched by GSEA. k Citrate cycle (TCA cycle) pathway enriched by GSEA. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001

    Article Snippet: Anti-cancer Metabolism Compound Library containing 237 anti-cancer metabolism compounds was purchased from TargetMol(L2130).

    Techniques:

    SDHC reprogramed fatty acid metabolism by regulating the PI3K/AKT pathways. a Western blotting was used to demonstrate the effects of silencing SDHC on the expression of critical enzymes involved in lipid metabolism, namely ACOX1, CPT1A, and ALDH3A2. b Western blotting was used to demonstrate the effects of overexpression SDHC on the expression of critical enzymes involved in lipid metabolism. c Migration assay analysis of orlistat in HCT116 and SW480 cells with SDHC decreased. d The effect of orlistat on liver metastasis formation in nude mice bearing HCT116 cells with SDHC decreased. e Western blotting reveals the levels of PI3K, p-PI3K, AKT and p-AKT in HCT116 and SW480 cells with SDHC decreased and control. f Western blotting demonstrates the levels of PI3K, p-PI3K, AKT and p-AKT in HCT116 and SW480 cells with SDHC overexpress and control. g Western blotting demonstrates the levels of AKT, p-AKT, ACOX1, CPT1A and ALDH3A2 in HCT116-sh-SDHC and SW480-sh-SDHC cells treated with MK-2206. h Migration assay analysis of MK-2206 in HCT116 and SW480 cells with SDHC decreased. i Schematic depiction of the regulation on fatty acid metabolism by SDHC in CRC cells. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001

    Journal: Journal of Translational Medicine

    Article Title: Deficiency of SDHC promotes metastasis by reprogramming fatty acid metabolism in colorectal cancer

    doi: 10.1186/s12967-024-05361-x

    Figure Lengend Snippet: SDHC reprogramed fatty acid metabolism by regulating the PI3K/AKT pathways. a Western blotting was used to demonstrate the effects of silencing SDHC on the expression of critical enzymes involved in lipid metabolism, namely ACOX1, CPT1A, and ALDH3A2. b Western blotting was used to demonstrate the effects of overexpression SDHC on the expression of critical enzymes involved in lipid metabolism. c Migration assay analysis of orlistat in HCT116 and SW480 cells with SDHC decreased. d The effect of orlistat on liver metastasis formation in nude mice bearing HCT116 cells with SDHC decreased. e Western blotting reveals the levels of PI3K, p-PI3K, AKT and p-AKT in HCT116 and SW480 cells with SDHC decreased and control. f Western blotting demonstrates the levels of PI3K, p-PI3K, AKT and p-AKT in HCT116 and SW480 cells with SDHC overexpress and control. g Western blotting demonstrates the levels of AKT, p-AKT, ACOX1, CPT1A and ALDH3A2 in HCT116-sh-SDHC and SW480-sh-SDHC cells treated with MK-2206. h Migration assay analysis of MK-2206 in HCT116 and SW480 cells with SDHC decreased. i Schematic depiction of the regulation on fatty acid metabolism by SDHC in CRC cells. Data are presented as the mean ± SD and analyzed by Student’s t-test. *P < 0.05, **P < 0.01, ***P < 0.001

    Article Snippet: Anti-cancer Metabolism Compound Library containing 237 anti-cancer metabolism compounds was purchased from TargetMol(L2130).

    Techniques: Western Blot, Expressing, Over Expression, Migration